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ROS also induces melanocytes to adopt a senescence-associated secretory phenotype (SASP), where they express pro-inflammatory cytokines that inhibit melanocyte proliferation and recruit immune cells to clear the “senescent melanocytes.” Furthermore, endoplasmic reticulum dysfunction in vitiligo patients exacerbates oxidative stress abnormalities and stimulates the secretion of IL-6 and IL-8, triggering immune responses

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A second mechanism by which GLY could affect glucose homeostasis lies in its potential ability to modulate insulin secretion from pancreatic beta-cells by activating ligand-gated chloride channels [78,81]

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